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The present review tends to articulate important information on SOD, CAT and GPX as first line defense antioxidant enzymes

Subsequent structural and biochemical studies revealed that, in the absence of its cofactor pyridoxal-5-phosphate (PLP) or substrate, SHMT2 forms an inactive dimer that binds BRISC complex and sterically blocks BRCC36, acting as an endogenous competitive inhibitor of BRISCs nonspecific deubiquitinating activity [131, 132] (Fig