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Some analyses were performed on computational resources from the Rockefeller University High Performance Computing Resource Center (RRID: SCR_025889)

106 LewerenzJ.MaherP

Postulated mechanisms of metformin action, including AMPK activation and complex I inhibition, have been demonstrated in the setting of a consistent pleiotropic activity of this drug, suggesting that several molecular targets could be involved (Patil et al., 2014

Taken together, these data show that although ABCC1 did not contribute to the acquired resistance to Venetoclax, inhibition of ABCC1 can restore Venetoclax sensitivity in resistant AML cells