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Within experimental models, it is commonly investigated for peptide-mediated signalling dynamics and molecular interaction mechanisms

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In response to oxidative challenges, Nrf2 dissociates from its inhibitor, Kelch-like ECH-associated protein 1 (Keap1), moves to the nucleus, and binds to the antioxidant response element (ARE) to activate the Nrf2-dependent antioxidant system

The spectrum of diseases involving mitochondrial dysfunction is a consequence of the multiple roles of mitochondria in metabolic and oxidative phosphorylation system (OXPHOS)-related processes 3

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