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The 3D models also predicted that the mutated FGF5 lacked the -12 strands that define the canonical trefoil of the FGF family (Fig

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Anemia is multifactorial, arising from defective CCO activity in erythrocyte precursors (normocytic anemia) and, in the case of TTM, from CP dysfunction, leading to iron retention and sideroblastic features [224, 244]

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That single design decision changes everything about how the molecule behaves: ACTH 4-7 itself is cleaved in seconds by brain and plasma peptidases, whereas Semax persists long enough to drive profound transcriptional effects on brain-derived neurotrophic factor and nerve growth factor