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However, under nutrient-rich conditions, mTORC1 phosphorylates TFEB, keeping it in the cytosol by anchoring it to 14-3-3 proteins, thus preventing it from activating its target genes ( Other related transcription factors, such as transcription factor binding to IGM enhancer 3 (TFE3) and microphthalmia-associated transcription factor (MITF), also contribute to this program and can compensate for TFEB when needed

After detoxification by glutathione (GSH) conjugation catalyzed by GSTs, the body can eliminate potentially harmful and toxic compounds

Phospholipid dynamics in ex vivo lung cancer and normal lung explants

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Summary: The adoption of these technologies and the establishment of novel therapeutic interventions is difficult since there is a gap in terms of a complete understanding of the pathophysiological mechanisms at the cellular and molecular level and the lack of data in terms of the assessment of safety and bioavailability differences in the individuals patients