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At pico molar concentrations, however, Hinge prevented HGF, Nle 1 -AngFV and Dihexa induced spinogenesis (Figure 12B-D) further suggesting that the effects observed for our angiotensin IV ligands are HGF/c-Met mediated

The presence of insulin resistance and elevated pro-inflammatory cytokines (e.g., tumor necrosis factor-) induce hepatic GH resistance (64), causing increases in GH and decreases in IGF-I levels ( Table 1 )

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