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Fig EV5A

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In spontaneously hypertensive rats, treatment with FAD inhibited pathological cardiac hypertrophy and fibrosis and these effects were associated with a significant increase in SCAD activity, higher ATP content, and a decrease in ROS level [174], thereby suggesting a cardiac protective effect of this compound that should be considered at least to hinder the progression of cardiovascular diseases to HF

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Mechanistically, studies suggest vascular and tendon/muscle healing effects