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Aside from their involvement in the regulation of peroxisome body proliferation in S

The ability of the GGT-null mouse to cleave the -glutamyl bond of LTC 4 and release LTD 4 led Carter and coworkers to initiate a search for another enzyme that could cleave -glutamyl bonds

While some of these may be associated with defense responses, further analysis is required to distinguish infection-specific responses from baseline genotypic differences

Hyperglutamylation upon loss of deglutamylases CCP1 or CCP5 leads to architectural defects of the axoneme and loss of the bulge region

SOD1 mutations associated with amyotrophic lateral sclerosis analysis of variant severity