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we take the regeneration rate from[10]

In turn, enhancing KCC2 activity not only potentiated L838,417-induced analgesia, it rescued its analgesic potential at high doses, revealing a novel strategy for analgesia in pathological pain, by combined targeting of the appropriate GABA A R-subtypes and restoring Cl - homeostasis

However, identifying patient subgroups that may potentially benefit from glibenclamide remains a critical area for future research

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Previous study established a LPS-induced murine sepsis model ( in vitro experiments demonstrated that MMPP inhibited cardiomyocyte death by attenuating oxidative stress and inflammatory responses while maintaining mitochondrial homeostasis