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4e, 5e)

However, when Cys 129 residue of PTPN22 is mutated, the disulfide bond cannot form and the enzyme is exposed by oxidation, resulting in inactivation (93)

The Th17/Th2 subset has been identified in mice, which act as a pro-inflammatory phenotype through the secretion of IL-17A (59)

Interestingly, with progressive increase in IOP, autophagy is initially activated in the dendrites of RGCs and protects cells, but its subsequent activation in the cytoplasm disrupts homeostasis and induces cell death 21,22,70,71,72

pylori infection, both the gastrointestinal lumen and gastric juice ascorbic acid content decrease