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Although much has been learned from targeted studies focused on single proteins [e.g., determination of SNARE binding domains assessed cleavage/deletion-mediated protein inactivation (Schiavo et al., a priori for functional and targeted studies

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This neuroimmune-tumor connection is also paradoxical as links also exists between chronic inflammation and tumorigenesis, with glioma cells producing pro-inflammatory cytokines, chemokines, and growth factors that create an inflammation-enriched TME (65, 66)

Hill, University of Louisville, USA

doi: 10.3390/ijms23031105