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In ALS, pathological intronic hexanucleotide GGGGCC expansions in the C9orf72 gene are thought to contribute to neurodegeneration by multiple mechanisms, including toxic cytoplasmic dipeptide repeat protein aggregates, toxic RNA gain-of-function, or loss of function due to haploinsufficiency [20, 21]

Conservative measures had not help so made the long trek from the midlands to seek the help from Dr Akhtar

Thus, regular supplementation accelerates fat metabolism

Finally, integrating circadian biology into the development of pharmacological modulators of UPS activity presents an exciting direction

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