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Servicio de Bioqumica Clnica, Hospital Hospital Universitari de Girona Josep Trueta

Therefore, we propose that latency and recurrent EBV reactivation could generate an acquired immunodeficiency syndrome in three steps: first, an acquired EBV immunodeficiency develops in individuals with weak EBV HLA-II haplotypes, which prevents the control of latency I cells

Curcumin-induced elevation of BDNF in this context may contribute to stabilization of dopaminergic neurotransmission and amelioration of motor deficits [11]

Cell Death Differ 6 : 10811086

Lewis KA, Tollefsbol TO (2016) Regulation of the telomerase reverse transcriptase subunit through epigenetic mechanisms