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The free (unbound) T3 is what can enter and affect the body tissues

In terms of mitochondrial dysfunction, the examples of UQCRC1 and CHCHD2 have suggested that PD is not just a cI disorder

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Interestingly, astrocytes (which protect neurons and promote sprouting) are activated because glial fibrillary acidic protein (GFAP) immunoreactivity increases in the striatum and indusium griseum upon METH treatment, thereby implying complex mechanisms involving guidance molecules and cytotoxic mediators in the neurotoxic consequences of METH ( FIGURE 4 Long-Term Neurotoxicity The effects of long-term exposure to METH are different from those of acute injury