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Control of excitatory CNS synaptogenesis by astrocyte-secreted proteins Hevin and SPARC

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This increase in OXPHOS drives parallel increases in ROS production and accumulation of mitochondrial damage, which is likely a cause for the observations that AML blasts have a reduced capacity to tolerate exogenous oxidative stress (34, 35)

[CrossRef] [PubMed] [Google Scholar] The effects of paracetamol on the liver and kidney functions of a rat model following prolonged alcohol administration