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Under ultrasound, it rapidly produced ROS to kill P

This beneficial effect was attributed to a plethora of mechanisms, including (i) improved enzymatic antioxidant system capacity with a concomitant decline in the level of lipid peroxidation, (ii) increase in activity of glutathione S-transferase, (iii) downregulation of glucose transporter 1 (GLUT1) restoring normal declined blood glucose levels in tumor cells, (iv) inhibition of lactate dehydrogenase dependent glycolytic machinery, (v) vasodilation of blood vessels resulting in decreased tumor hypoxia and downregulation of the angiogenesis markers

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Risk increases significantly when administered outside licensed clinical settings

doi: 10.1523/jneurosci.22-24-10662.2002 63 TangHYJiangAJMaJLWangFJShenGM