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While existing research has identified hyperuricemia involvement in numerous biological processes and signaling pathways, the precise mechanisms connecting elevated UA levels to disease etiology remain to be fully elucidated

NO exhibits well-characterized pleiotropic biological activities, including potent vasodilation, anti-inflammatory actions, modulation of ECM metabolism, and suppression of apoptosis

24 These substances promote inflammation as well as sensitize nociceptive pathways, leading to increased pain sensitivity, known as hyperalgesia, and pain from normally non-painful stimuli, termed allodynia

Stress management influences digestive function through the gut-brain axis

Treatment with RY-A significantly improved these pathological changes and attenuated the CCH-induced neural tissue damage