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When administered, Sermorelin binds to receptors on the pituitary gland, signaling it to release more HGH

To our knowledge, this is the first intervention to specifically target microglial lipid-metabolic dysfunction evoked by butyrate depletion, providing pre-clinical proof-of-concept for repurposing butyrate against MPs-triggered neurotoxicity and broadening its therapeutic scope in neurodegenerative diseases