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We can see a large number of articles using glutamate modeling to study the mechanism of excitotoxicity, most of which only look at the increase in calcium ions and mitochondrial dysfunction caused by excitotoxicity

In a study employing PC-12 cells, which are frequently used in neurobiological studies, the mechanism of toxicity revealed that Pb 2+ can trigger apoptosis through the altered ratio between the antiapoptotic protein BCl-2 and the proapoptotic proteins Bax and p53 in favor of their proapoptotic activity (Xu et al

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and diseases of the nervous system, including depression, dementia, Alzheimers, and Parkinsons diseases [80,81,82,83,84,85]

Role of pharmacokinetics and metabolism in drug discovery and development