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This critical comparison reveals a therapeutic paradox: broad inhibition of FAO might simultaneously affect both pro-tumoral and anti-tumoral cells, leading to complex and potentially unpredictable outcomes

Potential therapeutic targets The roles of TLRs in inflammation promote the development of the therapeutic potential of targeting TLRs in the treatment of atherosclerosis

Finally, these findings were replicated in younger children

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Microglial ferroptosis has been shown to amplify neurotoxicity by releasing pro-inflammatory mediators (such as TNF- and IL-1), and ferroptosis-related genes including ACSL4 and ALOX15 are highly enriched in microglia within PVL lesions