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One of the most critical mechanisms involves modulation of the reninangiotensin system (RAS), which serves as a vital link between gut microbiota dysbiosis and renal pathology (150, 151)

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Additionally, genes previously associated with a dedifferentiated state, such as Axl, Itga8 , Timp1 , Hmga1 , and Nkd2 , were upregulated on FN compared to COL I (Fig