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We demonstrate that acetaminophen treatment of Drosophila results in similar pathophysiologic alterations as those observed in mammalian systems, including a robust production of reactive oxygen species, depletion of glutathione, and dose-dependent mortality

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Notably, one area that has not been extensively addressed in the studies included in this review but is gaining attention in the broader literature is the integration of neurophysiological markers into VR-based cognitive training

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Parkinsonian syndrome and ataxia as a presenting finding of acquired hepatocerebral degeneration