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Disclosures: Michael Nudo: Nothing to Disclose, Felice Cinque: Nothing to Disclose, Sahar Saeed: Novo Nordisk: Consultant, Amanda Lupu: Nothing to Disclose, James Gazetas: Nothing to Disclose, Luz Ramos Ballesteros: Nothing to Disclose, Wesal Elgretli: Nothing to Disclose, Erica Moodie: Nothing to Disclose, Colleen Price: Nothing to Disclose, Ken Monteith: Nothing to Disclose, Curtis Cooper: Nothing to Disclose, Sharon Walmsley: Nothing to Disclose, Neora Pick: Nothing to Disclose, Melanie Murray: Nothing to Disclose, Nadine Kronfli: Nothing to Disclose, Joseph Cox: Nothing to Disclose, Cecilia Costiniuk: Nothing to Disclose, Alexandra De Pokomandy: Nothing to Disclose, Jean-Pierre Routy: Nothing to Disclose, Marina Klein: Nothing to Disclose, Bertrand Lebouch: Merck Canada: Speaking and Teaching, Gilead: Speaking and Teaching, ViiV healthcare: Consultant, Gilead: ASAP study managed by RI-MUHC, Merck: I score study and core set study managed by RI-MUHC, ViiV: PIVHOT progra mfro patients managed by RI-MUHC, Giada Sebastiani: Novo Nordisk: Consultant, Abbvie: Speaking and Teaching, Merck: Consultant, Gilead: Speaking and Teaching, Theratechnologies Inc.: Grant/Research Support 625 FREE FATTY ACID INDUCES APOPTOSIS VIA LNCRNA NEAT1/MIR-26A/PTEN AXIS IN HEPATIC CELLS Amrendra Kumar Sah 1 Teja Naveen Sata 1 Abdullah Mohit 1 Gopal Sharma 1 Md Ismail 1 Md Musa Hossain 1 Sarita Gupta 2 Sanal Madhusudana Girija 2 Senthil Venugopal 1 , 1 South Asian University, 2 Institute of Liver and Biliary Sciences Background: Metabolic dysfunction-associated steatotic liver disease (MASLD) is one of the leading causes of end stage chronic liver disease and HCC
ClinicalTrials.gov website
The first stage of the pathogenesis of CTD-ILD is inflammation and alveolar epithelial damage, which may be triggered by infection, by inhalation of irritating agents or by gastro-esophageal reflux disease in genetically susceptible individuals (3)
SK-MEL-103 model of melanoma For the melanoma experiments, 12-week-old NRG female mice were used
